Level D· Preclinical EvidenceAnimal Study

TEAD1 Enhances Exosome Secretion and Promotes Exosome-Mediated Tissue Regeneration.

Pu Y., Wan Y., Shi W., Li B., Zhang H., Wu J.

Animal Study on Diabetic Foot, Spinal Cord Injury, Chronic Wound, published in Adv Sci (Weinh) (2026) — summary generated from the PubMed abstract.

This page is generated from the PubMed record. The Thai description is an automated summary of bibliographic fields and the abstract, not a full translation, and is not medical advice.

Study type
Animal Study
Journal
Adv Sci (Weinh) (2026)
Country
Germany
Reported sample size
PMID
41815094
DOI
10.1002/advs.202514104

Abstract (original English)

Exosomes serve as intercellular communication vectors and are involved in a broad range of physiological functions. Although exosome-based therapies have demonstrated diverse functional potential, the regulatory mechanisms underlying their biogenesis and secretion remain poorly understood. Here, we report that TEAD1 functions as a molecular switch, dramatically enhancing the synthesis and secretion of exosomes. Mechanistically, TEAD1 enhances exosome secretion by promoting the expression of exosome secretion-associated proteins RAB11, CD9, and SNAP23. We found that TEAD1 enhances exosome secretion from adipose-derived mesenchymal stem cells, thereby promoting skin wound healing in diabetic mice. Similarly, TEAD1 promotes the release of exosomes from bone marrow-derived mesenchymal stem cells, thereby facilitating spinal cord injury (SCI) repair. Our study elucidates a novel role for TEAD1 in driving exosome secretion in different cell types, highlighting the therapeutic potential of TEAD1 in enhancing tissue regeneration, particularly in diabetic wound healing and SCI repair.

What this study does not prove

  • This study does not prove SVF is an approved treatment or a replacement for standard care.
  • This is preclinical work; animal or laboratory results cannot be applied to humans.

Evidence level

Evidence comes from animal or laboratory studies and has not been confirmed in humans.

How we grade evidence
AnimalsTEA Domain Transcription FactorsExosomesMiceWound HealingTranscription FactorsDNA-Binding ProteinsMesenchymal Stem CellsSpinal Cord InjuriesHumans

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